Autoimmune Neurological Conditions Associated With Zika Virus Infection

Título traducido de la contribución: Condiciones neurológicas autoinmunes asociadas con la infección por el virus Zika.

Yeny Acosta-Ampudia, Diana M Monsalve, Luis F Castillo-Medina, Yhojan Rodríguez, Yovana Pacheco, Susan Halstead, Hugh J Willison, Juan-Manuel Anaya, Carolina Ramírez-Santana

Resultado de la investigación: Tipos de Contribución a publicaciones especializadasArticulo

5 Citas (Scopus)

Resumen

Zika virus (ZIKV) is an emerging flavivirus rapidly spreading throughout the tropical Americas. Aedes mosquitoes is the principal way of transmission of the virus to humans. ZIKV can be spread by transplacental, perinatal, and body fluids. ZIKV infection is often asymptomatic and those with symptoms present minor illness after 3 to 12 days of incubation, characterized by a mild and self-limiting disease with low-grade fever, conjunctivitis, widespread pruritic maculopapular rash, arthralgia and myalgia. ZIKV has been linked to a number of central and peripheral nervous system injuries such as Guillain-Barré syndrome (GBS), transverse myelitis (TM), meningoencephalitis, ophthalmological manifestations, and other neurological complications. Nevertheless, mechanisms of host-pathogen neuro-immune interactions remain incompletely elucidated. This review provides a critical discussion about the possible mechanisms underlying the development of autoimmune neurological conditions associated with Zika virus infection.

Idioma originalEnglish
Páginas116
Número de páginas14
Volumen11
Publicación especializadaFrontiers in Molecular Neuroscience
DOI
EstadoPublished - abr 11 2018

Citar esto

Acosta-Ampudia, Yeny ; Monsalve, Diana M ; Castillo-Medina, Luis F ; Rodríguez, Yhojan ; Pacheco, Yovana ; Halstead, Susan ; Willison, Hugh J ; Anaya, Juan-Manuel ; Ramírez-Santana, Carolina. / Autoimmune Neurological Conditions Associated With Zika Virus Infection. En: Frontiers in Molecular Neuroscience. 2018 ; Vol. 11. pp. 116.
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abstract = "Zika virus (ZIKV) is an emerging flavivirus rapidly spreading throughout the tropical Americas. Aedes mosquitoes is the principal way of transmission of the virus to humans. ZIKV can be spread by transplacental, perinatal, and body fluids. ZIKV infection is often asymptomatic and those with symptoms present minor illness after 3 to 12 days of incubation, characterized by a mild and self-limiting disease with low-grade fever, conjunctivitis, widespread pruritic maculopapular rash, arthralgia and myalgia. ZIKV has been linked to a number of central and peripheral nervous system injuries such as Guillain-Barr{\'e} syndrome (GBS), transverse myelitis (TM), meningoencephalitis, ophthalmological manifestations, and other neurological complications. Nevertheless, mechanisms of host-pathogen neuro-immune interactions remain incompletely elucidated. This review provides a critical discussion about the possible mechanisms underlying the development of autoimmune neurological conditions associated with Zika virus infection.",
author = "Yeny Acosta-Ampudia and Monsalve, {Diana M} and Castillo-Medina, {Luis F} and Yhojan Rodr{\'i}guez and Yovana Pacheco and Susan Halstead and Willison, {Hugh J} and Juan-Manuel Anaya and Carolina Ram{\'i}rez-Santana",
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Autoimmune Neurological Conditions Associated With Zika Virus Infection. / Acosta-Ampudia, Yeny; Monsalve, Diana M; Castillo-Medina, Luis F; Rodríguez, Yhojan; Pacheco, Yovana; Halstead, Susan; Willison, Hugh J; Anaya, Juan-Manuel; Ramírez-Santana, Carolina.

En: Frontiers in Molecular Neuroscience, Vol. 11, 11.04.2018, p. 116.

Resultado de la investigación: Tipos de Contribución a publicaciones especializadasArticulo

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AU - Acosta-Ampudia, Yeny

AU - Monsalve, Diana M

AU - Castillo-Medina, Luis F

AU - Rodríguez, Yhojan

AU - Pacheco, Yovana

AU - Halstead, Susan

AU - Willison, Hugh J

AU - Anaya, Juan-Manuel

AU - Ramírez-Santana, Carolina

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N2 - Zika virus (ZIKV) is an emerging flavivirus rapidly spreading throughout the tropical Americas. Aedes mosquitoes is the principal way of transmission of the virus to humans. ZIKV can be spread by transplacental, perinatal, and body fluids. ZIKV infection is often asymptomatic and those with symptoms present minor illness after 3 to 12 days of incubation, characterized by a mild and self-limiting disease with low-grade fever, conjunctivitis, widespread pruritic maculopapular rash, arthralgia and myalgia. ZIKV has been linked to a number of central and peripheral nervous system injuries such as Guillain-Barré syndrome (GBS), transverse myelitis (TM), meningoencephalitis, ophthalmological manifestations, and other neurological complications. Nevertheless, mechanisms of host-pathogen neuro-immune interactions remain incompletely elucidated. This review provides a critical discussion about the possible mechanisms underlying the development of autoimmune neurological conditions associated with Zika virus infection.

AB - Zika virus (ZIKV) is an emerging flavivirus rapidly spreading throughout the tropical Americas. Aedes mosquitoes is the principal way of transmission of the virus to humans. ZIKV can be spread by transplacental, perinatal, and body fluids. ZIKV infection is often asymptomatic and those with symptoms present minor illness after 3 to 12 days of incubation, characterized by a mild and self-limiting disease with low-grade fever, conjunctivitis, widespread pruritic maculopapular rash, arthralgia and myalgia. ZIKV has been linked to a number of central and peripheral nervous system injuries such as Guillain-Barré syndrome (GBS), transverse myelitis (TM), meningoencephalitis, ophthalmological manifestations, and other neurological complications. Nevertheless, mechanisms of host-pathogen neuro-immune interactions remain incompletely elucidated. This review provides a critical discussion about the possible mechanisms underlying the development of autoimmune neurological conditions associated with Zika virus infection.

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