Abstract
A 39-year-old woman with recurrent severe anterior ischemic optic neuropathy in the setting of Sjogren's syndrome was found to have high-titer IgM anticardiolipin antibodies (aCL) that were enhanced by the phospholipid-binding glycoprotein β2GPI. The recognition of IgM aCL-associated vasculopathy as a possible etiologic factor in the optic neuropathy in Sjogren's syndrome may be relevant in deciding appropriate therapy.
Original language | English (US) |
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Pages (from-to) | 155-157 |
Number of pages | 3 |
Journal | Lupus |
Volume | 4 |
Issue number | 2 |
DOIs | |
State | Published - 1995 |
Externally published | Yes |
All Science Journal Classification (ASJC) codes
- Rheumatology